The bilateral GM Primary auditory cortex TE1.1 R, as defined in the Juelich maxprob thr25 2mm atlas, corresponds to a cytoarchitectonically distinct subregion of the human primary auditory cortex located on the transverse temporal gyrus (Heschl’s gyrus) of the superior temporal lobe. This area is part of the core auditory field, characterized by granular cortex receiving dense thalamocortical input from the medial geniculate body and exhibiting tonotopic organization that supports frequency-specific sound processing. TE1.1 forms one component of a broader primary auditory field complex (together with TE1.0 and TE1.2), contributing to early-stage analysis of acoustic features such as pitch and simple temporal patterns, and providing processed auditory information to surrounding belt and parabelt regions involved in higher-order auditory perception and integration. No direct Wikipedia article exists for TE1.1; a related structure is Primary auditory cortex.
The bilateral GM Primary auditory cortex TE1.1 R (Juelich maxprob thr25 2mm) has been implicated in several genetically influenced neuropsychiatric and sensory traits, although few genome-wide association studies (GWAS) target this subregion specifically; rather, it emerges within broader temporal or auditory cortex analyses. Polygenic effects involving genes related to synaptic plasticity, glutamatergic signaling, and neurodevelopment (for example, variants near GRIN2A, FOXP2, and CNTNAP2) have been associated with structural and functional differences in primary auditory and superior temporal cortices, which include regions overlapping TE1.1. GWAS of cortical surface area and thickness have identified loci (e.g., in genes such as MIR137, HMGA2, and DCDC2) that show significant associations with temporal lobe morphology encompassing primary auditory fields, linking this region to general neurodevelopmental architecture. Genetic studies of language, reading and dyslexia, stuttering, and schizophrenia have reported altered activity or structure in primary and association auditory cortices, suggesting that risk alleles affecting these traits may partly act through TE1.1-related circuits, though the associations are usually at the level of networks rather than this atlas parcel alone. In addition, variants associated with tinnitus, hyperacusis, and auditory hallucinations have been related to altered excitability and connectivity in primary auditory regions, again implicating TE1.1 as part of a genetically modulated auditory processing system rather than as an isolated target.
Overview generated by GPT-4o (2026).
Region ID: 44
Hemisphere: bilateral
Atlas: Juelich maxprob thr25 2mm
Full Quality Version: Download MP4
Full Quality Version: Download MP4
Wali Sidiqyar*, Gaurav Rudravaram*, Elyssa M. McMaster, Trent M. Schwartz, Adam M. Saunders, Kurt G. Schilling, Bennett A. Landman "Introducing SPINS: A Shared Public Visualization Library of Neuroanatomical Structures." Medical Imaging with Deep Learning- short paper
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